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Furthermore, Nrf2/HO-1 interacts with the NF-KB and JAK-STAT pathways in various contexts: Nrf2/HO-1 can reduce NF-KB phosphorylation and thereby dampen inflammation, whereas proinflammatory cytokines such as TNF- and IL-6, released following NF-KB activation, upregulate HO-1 in a feedback loop (125, 129)

This suggests that fast-twitch fibers are more susceptible to damage induced by oxidative stress (69)

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This pattern likely reflects broader flaws in the reporting standards of preclinical animal research rather than issues specific to the included publications