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GHK-Cu suppresses inflammation through multiple signaling pathway interventions, reducing production of pro-inflammatory cytokines like TNF-alpha and IL-6 while decreasing inflammatory cell infiltration into tissues

(3) This theorized pathway involves the activation of phospholipase C (PLC), which some researchers believe may lead to the increased release of inositol triphosphate (IP3) and diacylglycerol (DAG)

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