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More importantly, IR R-1 clones exhibited higher basal levels of DNA damage and repair proteins in the p53 (low p21 expression), DDR (H2AX and pChk2), NHEJ (Ku80), and HRR (Rad51) pathways than did U87-MG cells (Fig

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In serum, first-episode, antipsychotic-nave patients show reductions in TRP, KYN, KYNA, anthranilic acid, and modestly lower 3-HK and QA, indicating early KP dysregulation, and long-term antipsychotic treatment partially normalizes these metabolites [238]