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Research demonstrates that GHK-Cu stimulation of collagen synthesis begins at concentrations between 10 and 10 M and maximizes at 10 M, with effects independent of changes in cell proliferation, indicating direct activation of synthetic machinery rather than simply increasing fibroblast numbers

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however, this new state is often described as intermediate, distinct from both the pre-treatment CF state and that of healthy controls, with evidence of incomplete resolution of neutrophilic inflammation (Maher et al., 2024)

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