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The lack of success at Phase III was attributed to factors such as high baseline immunosuppressive therapy in patients and complex disease mechanisms in SLE, potentially limiting the application of Rituximab as a monotherapy in SLE (119)

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Supplementation with farnesyl pyrophosphate, a product of CoQ10 synthesis, has been shown to inhibit ferroptosis induced by FIN56 (67), highlighting the endogenous inhibitory role of CoQ10 in this process