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As a result, whereas in most somatic cells G6PD is subject to turnover, in red blood cells any G6PD molecule undergoing denaturation or proteolytic breakdown cannot be replaced (this condition is true, of course, not only for G6PD but also for most other red blood cell enzymes )

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Moreover, they also have a positive effect on cardiometabolic risk factors most probably translating to a moderate reduction in the risk of major adverse cardiovascular events that still remain undetermined

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