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Based on the microglia transplantation approaches described (Table 2), it is appealing to hypothesize that bone marrow or hematopoietic cells could be modified ex vivo to express factors important for modulating an inflammatory response (such as IL-4, IGF-1, BDNF, GDNF) and/or blocking TLR/CD14 and then transplanted into ALS patients, taking advantage of the cells ability to migrate in the CNS (Fig

Energy deficiency can stimulate the proliferation of mitochondria of smooth muscle and small vascular endothelial cells and cause angiopathy (such as MELAS syndrome), resulting in damage to microvascular hemoperfusion and insufficient energy supply to the brain (16)