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New insight into glucose metabolism dysregulation in TBI Current consensus suggests that primary TBI immediately resulting from mechanical stress and displacement of brain tissues is compounded by progressive secondary injury (damage to the BBB, protein aggregation, etc.) that is partially mediated by metabolic dysfunction [27]

The lip tissue is particularly vulnerable to pigmentation changes because it is thin, constantly exposed to UV radiation, and rich in melanocytes that respond quickly to external stimuli

Existing evidence suggests that, even within the conventionally defined normal range, relatively higher TG levels are associated with IR and with an increased future risk of T2DM (103, 104)

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