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The pSDU1-I-Sce I plasmid in mutant cells was eliminated by spontaneous loss, as described earlier (Wu et al., 2.9

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Pertinent to epilepsy, mice lacking mitochondrial SOD ( Sod2/ ) and heterozygous animals ( Sod2+/ ) show higher seizure susceptibility and concomitant degeneration, introducing the notion that O 2 are key players in the long-term brain changes after a seizure episode [22,23]

doi:10.3390/nu8020068 Drake VJ