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In addition, CaMKII translocates from dendritic spines to inhibitory synapses upon weak chemical stimulation by NMDA and promotes the insertion of GABA A Rs to inhibitory synapses and enhancement of inhibitory transmission (Marsden et al., 2014) found that the chemical inhibitory long term potentiation (iLTP) triggered by NMDA application (up to 30 min) requires CaMKII-dependent phosphorylation of GABA A R subunit 3 at serine 383 that in turn promotes the synaptic recruitment of gephyrin from extrasynaptic sites

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