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It was proven that patients with early carnitine treatment and without frequent metabolic decompensations were less vulnerable to the development of pathological, neurological, and neuropsychological attributes, including ASD, in research concerning eight propionic acidosis patients with autistic symptoms (e.g., repeated movements or hand fluttering, stereotypic behavior, disturbances in social interactions, difficulty in communication with other people), who were treated with L-at 50 mg/kg/daily, biotin at 10 mg/day, and a low-protein diet [131]

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The subcutaneous approach here is this sites framing, not the established route [4]

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