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This is especially important for people with active cancer, a history of cancer, or anyone receiving cancer treatment, since NACs relationship with cancer biology is complicated

This interaction affects the expression of all the downstream targets causing significant metabolic depression, low cellular ATP content and, then, lipid accumulation ( FIGURE 2 Therapeutic Interventions for Rescuing Fatty Acid Oxidation As described above, dysfunctions of the mitochondrial -oxidation trigger kidney injury, therefore, mitochondria represent a potential drug target

10.1016/j.bbamcr.2015.12.017 Biochim

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