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And CD4 + CD25 + T cells effectively inhibited colonic effector T cell activity and the production of Th1 (interferon (IFN)-, IL-2) and Th2 (IL-5, IL-13) cytokines in vitro, while the frequency of Treg cells in the inflammatory area increases with the increase of active UC disease activity [68, 69]

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In this study, the authors showed that superoxide induced by DOCA-salt treatment caused increased vascular hydrogen peroxide production, which was significantly reduced by the eNOS inhibitor N G -nitro-L-arginine methyl ester