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Mechanistically, this may relate to GHK-Cu's ability to upregulate matrix metalloproteinases (MMP-1, MMP-2), enzymes that can fragment collagen if their activity exceeds the body's ability to rebuild
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Because NO can suppress IDO expression and promote its degradation [166], the NO-scavenging activity of 3-HAA may also indirectly sustain KP flux, highlighting the complex feedback loops linking inflammation, redox balance, and TRP metabolism in MDD
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