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Simultaneously, prolactin can alter the expression of lipidogenesis genes in subcutaneous white adipose tissue and exacerbate glucose intolerance induced by a high-fat diet ( 4.3 Insulin resistance and glucose metabolic dysregulation Clinical, epidemiological, and experimental studies have consistently shown that hyperprolactinemia is strongly associated with glucose metabolism disorders that primarily manifest as insulin resistance and impaired glucose tolerance

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This antioxidant activity is largely attributed to the presence of antioxidant molecules and trace elements, including zinc, IGF-1, and VEGF [86, 87]

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