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The purpose of this review is to provide a state-of-the-art interdisciplinary and translational approach to arterial stiffness, highlighting unique pathophysiological mechanisms (e.g., perivascular adipose tissue, extracellular vesicles), clinical relevance, and future directions

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36 Thus it appears that the mechanism of action of POL and STS is to produce endothelial damage, 37 but not thrombus formation associated with platelet aggregation