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This is a meaningful evidence gap

These results suggest that BPC157 was rapidly metabolized into low levels of a variety of small peptide fragments, finally resulting in a single amino acid represented by [ 3 H]proline, which entered the normal amino acid metabolism and excretion pathway in the body

Animal studies and clinical trials have demonstrated its potential to improve cognitive function, reduce oxidative stress, and protect against neurodegeneration

Nitric Oxide Mechanism in the Protective Effect of Antidepressants against 3-nitropropionic Acid-Induced Cognitive Deficit, Glutathione and Mitochondrial Alterations in Animal Model of Huntington's Disease