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Preventing this decrease, with the administration of a SIRT-1 activator, reversed ROS bursts, increased downstream nuclear PGC-1a and Nrf2 expression, and attenuated increases in mitochondrial membrane potential and neuronal apoptosis induced by ischemia/reperfusion injury [78]

3.5 Protein phosphorylation Protein phosphorylation, a critical post-translational modification, plays a pivotal role in the pathogenesis of PCOS by modulating signal transduction, enzyme activity, and cellular functions

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Myeloid programs further converge on lactylation-dependent immunosuppression: tumor signals activate PERKATF4 to induce GLUT1-high glycolysis in macrophages, generating lactate that promotes H3K18la at the IL10 promoter and robust IL-10 production