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Furthermore, ROS accumulation over time leads to irreversible damage, and Harman's (84) theory provides further insight into the role of extracellular ROS in triggering age-related cancer progression (85)

2 For example, IGF-1R-mediated activation of AKT, in turn, phosphorylates and inhibits pro-apoptotic proteins such as Bad and caspase 9 to promote survival

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