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The corresponding cystein and glutamate residues have been described as key catalytic residues of the aldehyde dehydrogenase family (Shortall et al., 2021)

Specifically, GPx1 deficiency augmented TNF suppression of the dual specific phosphatase DUSP4 to promote ERK1/2 activation, linking GPX1 and oxidant stress to MAPK regulation [6]

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