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Any of these make an auto-immune cause of B12 deficiency (PA, also called autoimmune gastritis 3 ) more likely

At the cellular level, B12 acts as a cofactor for two critical enzymatic reactions: Methionine synthase converts homocysteine to methionine, essential for DNA methylation and synthesis Methylmalonyl-CoA mutase involved in fatty acid metabolism and myelin production By restoring adequate B12 levels, injections enable normal red blood cell maturation in the bone marrow, correcting the megaloblastic anaemia

What GHK-Cu does at the cellular level The science gets interesting here

Other peptide-based medications are widely used for conditions like diabetes, multiple sclerosis, and cancer