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For example, in human PAECs under hypoxic conditions, the levels of ROS were increased, while the levels of mitochondrial thioredoxin 2, an antioxidant protein, were reduced which could contribute to exacerbating hypoxia-induced ROS production [157]

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AQ undergoes rapid and extensive hepatic metabolism to its pharmacologically active metabolite DEAQ, predominantly by cytochrome P450 2C8 (CYP2C8) (Li et al., 2002)