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Acknowledgements This research was supported in part by the National Institute of Health (R01-GM115622, R01-CA250503 and R01-CA268518 to J.W.), the Cancer Prevention & Research Institute of Texas (CPRIT, RP220480 to J.W.), and Michael E

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ApoE in Atherosclerosis In addition to apoA-I and HDL, the endogenous production of apoE by macrophages is critical in preventing atherosclerotic lesion formation

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