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In childhood SAM, immune function is repressed, aggravated by micronutrient deficiencies and resulting in sustained, low-grade inflammation, long-term metabolic damage and increased odds of infections and mortality [51]

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As glycolysis is the only source of ATP in the mature erythrocyte, genetic defects that affect either the level or activity of any of the glycolytic enzymes have significant effects on ATP production and therefore on erythrocyte survival

The expression of several GSTFs including GSTF7 and a GSTL gene were also markedly up-regulated after the fungal inoculation (Figure 2A)