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The growing understanding of AD as a multifactorial disease necessitates an approach that simultaneously addresses tau pathology, neuroinflammation, oxidative stress, mitochondrial dysfunction, and synaptic loss ( 4.2 The role of neuroinflammation in AD pathogenesis Recent evidence underscores neuroinflammation as a central player in AD progression

The same people experienced decreases in oxidative stress and increases in glutathione levels after taking antihypertensive medication for 3 months

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