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Endogenous toxins include the metabolic byproducts of our own cellular respiration, such as reactive oxygen species (ROS) and free radicals

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Based on the current evidence, we propose that the iron-overloaded, ROS-prone milieu of EMS renders EESCs susceptible to Erastin-triggered, multi-pathway regulated cell death that integrates ferroptosis with necroptotic and pyroptotic signaling, while ferritin handling and mitophagy act as key modulators of susceptibility

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