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Renal Cys levels have been utilized as an indirect indicator of efflux since the majority of GSH transported from the sinusoidal membrane of the liver into the plasma ends up being degraded in the kidney by the enzyme -glutamyl transpeptidase resulting in the release of Cys [60]

Acetaminophen overdose: N-acetylcysteine, administered either intravenously or orally, to restore glutathione levels Activated charcoal to absorb excess drug If acute liver failure occurs, intensive supportive therapy or liver transplantation may be required NSAID toxicity: Gastroprotective agents to prevent or treat gastric ulcers (eg, proton pump inhibitors [eg, omeprazole]) What are Emerging Therapies for Acetaminophen and NSAID Toxicity

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