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Understanding these mechanisms broadens the understanding of the consequences of pigmentation disorders but also paves the way for innovative therapeutic approaches aimed at reducing oxidative stress in melanocytes

Genetic variants that reduce UGT1A1 activity can slow glucuronidation and potentially prolong the negative effects on the endocrine system and liver

Agonistic analogs of growth hormone releasing hormone (GHRH) promote wound healing by stimulating the proliferation and survival of human dermal fibroblasts through ERK and AKT pathways