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The foundational mechanism paper that established MOTS-c as a therapeutically relevant peptide is also worth citing directly: a peer-reviewed Cell Metabolism study by Lee and colleagues showing that the mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance through the AMPK pathway

Elevated redox stress has been closely associated with a variety of clinical complications, including metabolic syndrome, pulmonary dysfunction, cardiovascular disease, and neurological disorders, all of which are commonly reported in both severe COVID-19 and PCC 26

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