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These two peptides are also being researched in the contexts of injuries to the brain, stroke, ALS, and Alzheimers disease, among other conditions

Taken together, these observations suggest that except in vitamin B 12 deficiency (which only responds to cyanocobalamin treatment because of the inactivation of methionine synthase), folate treatment should be the most adequate therapy to reverse hyperhomocysteinemia detected both under basal conditions and after an oral methionine load

The combination of dysregulated lipid homeostasis and impaired purine metabolism may offer a mechanistic explanation for the similar symptoms observed in ME/CFS and GWS

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