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Current literature suggests that the L-carnitine system is perturbed in AD, with multiple studies showing that proteins and metabolites related to the L-carnitine system are altered in the brain, CSF, and plasma of AD patients (Makar et al., 1995

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FAO not only supports ATP production but also promotes tumor progression, metastasis, and therapeutic resistance by regulating ROS levels, lipid metabolism-related transcription factors such as peroxisome proliferator-activated receptors (PPARs), and components of the AMP-activated protein kinase (AMPK) signaling axis [15, 16]