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In addition, compound YY-23 targets prefrontal cortical GABAergic interneuron NR2D-NMDARs, suppressing inhibitory GABAergic transmission while boosting excitatory signals to exert rapid antidepressant effects [43]

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In another large, open-label experiment that compared NAC to a controlled drug, patients using NAC had less deterioration of lung function as determined by forced expiratory volume in one second (FEV) [44]

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