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Perivascular inflammation may develop around damaged vessels, further impairing blood-brain barrier integrity and vascular function

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These data indicate that melatonin-dependent NRF2 activation and glutathione-mediated suppression of effector function are conserved between murine and human ILC2s

These neuroregeneration properties position Dihexa as a promising research compound for investigating brain repair mechanisms, developing treatments for degenerative neurological conditions, and understanding the potential for functional recovery following various forms of neural damage