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Extrinsically, dietary long-chain fatty acids (LCFAs) promote Th17 cells and exacerbate the pathogenesis of experimental autoimmune encephalomyelitis (EAE) ( Figure 1 )

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We also determined the relative importance of Dpp4 expression within endothelial, hematopoietic, and gut epithelial cells for control of lipid metabolism in mice fed regular chow (RC) or high-fat diets (HFD)