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(Article in Japanese) Mizuguchi T
In addition, DF-1 cells tried to repair the imbalance of the redox state due to the intense oxidative stress response triggered by viral infection by moderately increasing GSH concentration in host cells to mitigate the onset of apoptosis and maintain the replicative environment and persistence of IBDV, this phenomenon was also found in HCV infection of Huh7.5 cells (Anticoli et al
Role of human glutathione S-transferases in the inactivation of reactive metabolites of clozapine
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