slc25a39 glutathione DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Autoregulatory control of mitochondrial glutathione

slc25a39 glutathione

slc25a39 glutathione DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Autoregulatory control of mitochondrial glutathione

Name: slc25a39 glutathione DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Autoregulatory control of mitochondrial glutathione
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SKU: 17291426151

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This article is part of the PlexusDx Education Hub your resource for evidence-based guidance on blood testing & biomarkers

It was developed by Novo Nordisk under the code NNC 26-0161 and taken into clinical study for postoperative ileus, where gastrointestinal motility rather than growth hormone was the endpoint of interest