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Beyond deacetylation: the non-deacetylase functions of HDACs In addition to the protein deacetylation for HDACs, there are also several evidences showing that HDACs possess deacetylase-independent functions such as sumoylation, decrotonylation, ubiquitin-dependent function and lactylation eraser as shown below

Outcomes from other smaller trials (reviewed in 89) have suggested either a lack of an effect or the possibility of an increased risk of cancer

For example, the autophagy regulatory factor TP53INP2 can effectively promote the mitochondrial autophagy process, thereby reducing the accumulation of ROS within the cells and ultimately improving the related pathological phenotypes associated with sarcopenia [42]

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