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R-Baclofen Reverses Cognitive Deficits and Improves Social Interactions in Two Lines of 16p11.2 Deletion Mice

GSH deficiency compromised the activation of mammalian target of rapamycin-1 (mTOR) and expression of NFAT and Myc transcription factors, abrogating the energy utilization and Myc dependent metabolic reprogramming that allows activated T cells to switch to glycolysis and glutaminolysis [108]

The potential mechanism involves exerting a protective effect by increasing IL-10 levels and blocking the AMPK/mTOR signaling pathway