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Description

One mechanism involves reducing transforming growth factor beta-1 secretion in fibroblasts

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Preclinical studies report faster healing timelines, with tendon strength recovery observed within 14 to 28 days in controlled animal models

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Because individual peptides differ in their stability characteristics, no single reconstitution protocol is appropriate for every compound

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Research in aging mouse models shows that MOTS-c administration suppresses SASP markers and reduces the burden of senescent cells in metabolically active tissues [2]

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