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The compounds ability to activate the Nrf2-mediated antioxidant response helps mitigate the collateral damage caused by radiation and chemotherapy, particularly in highly vulnerable tissues such as hematopoietic systems and mucosal barriers ( 5.2 Clinical evidence and trial outcomes The translation of SFN from preclinical models to human clinical applications has generated substantial evidence supporting its potential in cancer prevention and management

Another proposed mechanism involves the binding of homocysteine to the collagenous matrix of bone, which may modify collagen properties and reduce bone strength (reviewed in 113)