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Research shows that its etiology (underlying cause) involves a mix of genetic factors, autoimmune activity, and environmental triggers such as stress or illness

A critical analysis of recent literature highlights the importance of integrative and cautious approaches that account for the developmental plasticity of the pediatric organism and the possible pleiotropic effects of early pharmacological intervention

The third molecular mechanism of PWS is less common and is found in about 3% patients with PWS, termed imprinting defect (ID), including epimutation and the PWS imprinting center (PWS-IC) deletion

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