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Eur J Immunol 31:35043512

The authors demonstrate that GLP-1R agonist treatment induces expression of tyrosine kinase IGF1R/ErbB (EGFR) pathways, however, agonist treatment can still increase intestinal growth in the absence of intestinal epithelial IGF1 receptor as well as EGF receptor signaling (Koehler et al., 2015)

The downstream effects include activation of hormone-sensitive lipase in adipose tissue (which breaks down stored triglycerides into free fatty acids), increased protein synthesis in skeletal muscle, enhanced cellular repair and turnover, elevated IGF-1 production from the liver, and improved lipid metabolism including reduced triglycerides