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Lysine-specific methyltransferase 2B (KMT2B), which belongs to the histone H3 lysine 4 (H3K4) methyltransferase family, accelerates the transcription of riboflavin kinase (RFK) by enhancing H3 methylation levels, thereby activating the TNF-/NOX2 pathway and promoting ferroptosis induced by myocardial ischemia-reperfusion[149]

Together, these findings highlight that both pharmacological antidepressants and bioactive dietary compounds like flavonoids can shift KP metabolism toward neuroprotective pathways, offering potential avenues to alleviate inflammation-driven depressive pathology

Used in preclinical research contexts : Discussed in literature mainly in in-vitro and animal models, not as validated clinical outcomes