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Impaired vascular perfusion at the microvascular level results not only in subcutaneous fluid build-up in the tissues, but also an accumulation of pro-inflammatory substances which have been attributed to the continuous nociceptor activation and related pathophysiological states including central nervous system sensitization and neuroinflammation
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We did not find a statistically significant change in calcium deposition or ALP activity caused by nicotine alone, but there was a decreasing effect on type I collagen production and MSC proliferation
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