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Similar content being viewed by others Background Both caffeine assumption and cigarette smoking are well known to yield considerable changes in cardiovascular hemodynamics

First, H-MAGMA 27 computes gene-based association statistics by aggregating SNP associations based on physical proximity to the target gene(s) measured by chromatin interaction maps from human brain tissue

[17] found that transcriptional factors Snail1 and Twist were upregulation and nuclear translocation in smokers and current-smoking COPD, and their expression is closely associated with EMT activity (S100A4 expression) and the levels of airflow obstruction