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Targeting Sema4D alleviates CNV progression and enhance anti-VEGF therapy To explore whether T cell-expressed Sema4D contributes to the progression of nvAMD and might affect the responsiveness to anti-VEGF therapy, we analyzed the expression of Sema4D on CD8 + T cells from the blood samples of nvAMD patients who received 3 + PRN anti-VEGF therapy

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Clinically, Th17 cells are key immune mediators of RA, these cells and their cytokine IL-17 are enhanced in circulation, synovium, and synovial fluid (42)